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Tolbert TM, Schoenholz RL, Parekh A, et al

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In pancreatic cells, GLP-1R activation signals primarily through G proteinmediated signaling pathways involving cyclic adenosine monophosphate (cAMP), protein kinase A ( PKA ), and cAMP response elementbinding protein ( CREB ), thereby enhancing glucose-dependent insulin secretion and supporting -cell function

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Following centrifugation, the supernatant was vacuum-dried

In same model, AHG elevated phosphorylation levels of insulin receptor substrate-1(IRS1), Akt, and AMPK levels that were decreased by HFD treatment (Chen Y