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To determine if ABT influences the transport kinetics of OTA, the same intravital experiment was performed after pre-treatment with ABT (Supplement Video 1B

Mitochondrial dysfunction and oxidative stress in Alzheimers disease: Implications for mitochondria-targeted antioxidant therapeutics

Furthermore, we confirm that the initial glutathionylation of the glutaredoxin moiety and not the glutathione transfer to the roGFP2 moiety is rate-limiting for the oxidation of fusion constructs at physiological or low micromolar GSSG concentrations, thus allowing glutaredoxin structure-function analysis in vivo

Lancet 384 (9945), 766781

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It can do so while also suppressing the release of glucagon, a hormone that raises blood sugar levels